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THE ENDO FILES
October 2013
Issue No.2
IN THIS ISSUE
Tooth #2: 
Procedural complication during endodontics  

    

It all began as a routine #2.  The patient was originally sent for consultation regarding discomfort in the upper right quadrant. Her chief complaint included"pain when chewing and a relatively constant ache" in the region. A quick intraoral examination revealed that the patient had an existing 2-x-x-5 bridge in the region, and she pointed to the area of #2 when prompted for the "trouble tooth".


The radiograph failed to clearly demonstrate any pathology. Except for calcification of the canals, the only finding of note was a lip on the M aspect of the crown that extended gingivally. My initial thought was that this lip might be causing a periodontal issue and that her pain was thus periodontal in nature. However, testing revealed a lack of response to cold and clear tenderness to percussion and palpation. It was clear to me that #2 had become necrotic and would require RCT.


From what I could gather from the radiograph, the tooth appeared to have two roots/canals. I followed my usual strategy with calcified upper molars, which is to find the palatal canal first, begin instrumentation, and then "take what the tooth gives me" (which in this case wasn't much!!). This strategy allows for proper re-orientation throughout the procedure, as canal locations can often surprise you in heavily calcified cases.


After locating the palatal canal, I traced the change in pulpal floor coloration toward the B (a good way to stay safe when locating calcified orifices). Eventually I located a second canal, which turned out to be the DB, but after enlarging this canal it became apparent that my guess of two canals proved to be incorrect.


And this is where things started to get interesting. Finding the remaining canals was proving to be extremely difficult. After several minutes, I located what I believed was the singular and final MB canal. However, enlargement of this canal rendered it palatal to the DB, indicating instead that it was MB2. Typically, I'm happy to find MB2; however, finding MB2 more easily than MB1 is a different ballgame! It can only mean that nature has done one heck of a job calcifying that main M canal.


I used MB2 as my reference point and started troughing buccally. And after a few swipes of the bur, I found a stick with my DG16, but unfortunately, this turned out to be the margin of the crown. A file xray verified that the file was indeed slipping between the margin of the tooth and the aforementioned M lip of the crown. Realizing this was the case, a few more swipes of the bur apically and toward MB2 finally revealed the sneaky MB1. However,now that all canals had finally been located, I had a new problem on my hands.


I wanted to seal the newly-open margin on the crown before proceeding with treatment, so as to prevent a hypochlorite accident, but in doing so, things became a little hairy. I grabbed the Geristore, a great glass ionomer that can be used for root repairs and began to prep the area for application. I then blew air out of the air/water syringe to dry the open margin. It was at this moment that the patient mentioned slight discomfort in the area above the tooth. I immediately stopped and asked her is everything was ok; she said yes, that the discomfort had gone away and that most likely she was simply sore from the grueling procedure. Seeing this as a plausible explanation, I continued and finished applying the Geristore.


Once the Geristore had set, I turned my attention back to the root canal at hand. However, out of the corner of my eye, I noticed something that caught me by complete surprise. Suddenly and surprisingly, the patient's right eye had swollen almost completely shut!!! I immediately dried the canals, placed calcium hydroxide, and sealed the access with a cotton pellet and Cavit. Removing the rubber dam, I began to examine the patient's eye as well as the cheek area beneath it and B gingiva intraorally.

 

                                                 
SUBCUTANEOUS AIR EMPHYSEMA

    

There were four key findings that allowed me to ascertain what had occurred and to thereby rule out a hypochlorite accident as the cause of this rapid eye swelling: 1) the fact that the patient had reported slight discomfort almost concomitant with me blowing air in the tooth; 2) the fact that the pain had subsided so quickly, especially without the help of saline/anesthetic irrigation; 3) although the eye was swollen, there was no discoloration, tenderness, or warm sensation in the associated soft tissues (a common finding in hypochlorite accidents), and 4) there was crepitus upon palpation of the swelling.

 

Instead, I was dealing with a rare, yet dangerous, endodontic procedural complication known as SUBCUTANEOUS AIR EMPHYSEMA. This phenomenon is caused by invasion of  compressed air into soft tissues through a disrupted intraoral barrier (in this case the open margin directly over the PDL), and it has been shown to occur during procedures spanning from surgical interventions such as extractions and implants to less invasive procedures such as root canals and even restorative dentistry. Often, gaseous invasion can be restricted to the connective tissues immediately adjacent to the entry site, but as with my case, air can spread between fascial planes and cause sudden swelling elsewhere (such as the eye with our patient). ***This is important to keep in mind especially when working on the lower, as the airway can quickly become compromised!

 

Treatment of mild to moderate cases of subcutaneous emphysema consists of frequent observation/follow-up and reassurance of the patient. In the majority of cases, the condition will improve within 2-3 days, although residual swelling can be present for up to 14 days and it is important to note that cervical emphysema usually takes longer to subside than facial emphysema. Prophylactic administration of antibiotics, preferably Augmentin, is also recommended to prevent secondary infection of distant soft tissue sites by oral flora traveling through the emphysematous tracts. In more severe cases, or when swelling appears to be worsening, immediate medical attention is mandatory, as tracheostomy may even become necessary in the most extreme situations.

 

In this particular case, management consisted of the following: 1) local I&D of the swollen area INTRAorally (to allow for diffusion of the gas outward); 2) prophylactic administration of Benadryl to rule out an allergic reaction; 3) prophylactic antibiotics; and 4) frequent follow-up. Luckily, our patient improved steadily and was back as good as new within a couple of days - even returning to finish the root canal 2 weeks later - but not before serving as a scary reminder of one of the many potential complications of endodontic treatment and dentistry in general. HECK, I  KNOW I LEARNED MY LESSON WITH POSITIVE AIR PRESSURE!!!

 

-Oscar


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THE ENDO FILES ARE INTENDED TO BE AN INFORMAL, INFORMATIVE AND POSSIBLY ENTERTAINING ACCOUNT OF CASES SELECTED BY DRS GARCIA AND PEGUERO.  SOME CASES WERE SUCCESSFUL, AND OTHERS NOT.  HOWEVER EVERY CASE SELECTED WILL HOPEFULLY OFFER THE READER A PEAK INTO THE MIND OF THE ENDODONTIST WHEN THE "GUTTAPERCHA HITS THE FAN"!  
WE HOPE YOU LEARNED SOMETHING....WE DID!

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