|
Annual Patient Workshop
Save the date:
Sunday
February 17, 2013
Hilton Dallas-Fort Worth Lakes Executive Conference Center | |
|
|
Therapy Tip
A reduced carbohydrate diet (especially refined sugars) is recommended for patients with Th1/Th17 inflammation because all carbohydrates are metabolized as sugars and L-form bacteria use sugars for 'fuel' to reproduce. Normal weight is an essential aspect of health. If weight is not within the normal body mass index (BMI) category, a low carbohydrate diet can facilitate weight loss.
| |
|
Recovery Reports
We are contacted daily by people with chronic illnesses who are looking for an effective treatment. Many ask us to provide evidence of efficacy in the form of statistics or stories. If you have recovered your health or had significant symptom improvement with Inflammation Therapy (or a similar treatment), please help us 'pay it forward' by telling your story. We will post it in the public section of our website to encourage others. Any report, short or long, with or without objective data (e.g., lab results, imaging reports) would be helpful. Please
send your story to
our email .
Thank you!
To see the latest recovery reports,
click here. | |
|
CIR Library Access
Access to our free, extensive, easy-to-read Library of Information
(see this sample page)
and Physicians' Reference Library is available to anyone, without enrollment in our counseling program. If you're interested in using this resource, please send a request to our email
address along with your doctor's name and fax number (in the US or Canada) or his/her email address, so we can notify your doctor that you have access to this information.
A list of the articles in our libraries is available at this link.
Physicians may use CIR libraries even if they don't have a patient enrolled in our counseling program. Interested medical practitioners should contact CIR and ask to register. | |
|
Survey
Volunteers who have a diagnosis of autoimmune or inflammatory disease or are chronically ill but who are not being treated with Inflammation Therapy, the Marshall Protocol or the Stillpoint Protocol are needed to be in the control group of our long-term clinical study. If you meet these criteria and would like to take part in this project (which will only take a few minutes
each month), please
contact us at our
email address. | |
|
CIR is an IRS-recognized 501(c)3 non-profit charitable organization.
Donate to CIR
in support of our educational and research efforts.
| |
|
Contact Us | |
| |
you may phone us toll free from anywhere in the US and Canada
1-888-846-2474
Skype
Chronic.Illness.Recovery
|
|
Have you enjoyed this newsletter?
| |
| |
|
|
HONcode
The Internet has become an important communication tool but it isn't always easy to tell which information is reliable. When it comes to your health, it's important to be sure you're accessing a credible source. Health On the Net Foundation has certified that the Chronic Illness Recovery website and forum conform to the principles of the HONcode for the dissemination of trustworthy health information for patients and professionals. The HON seal on our website is your assurance that we're providing medical information
you can trust.
Click here
for details.
| | |
|
|
|
 |
|
Greetings!
We are happy to announce that our third annual CIR Patient Workshop will be held at the Hilton Executive Conference Center in Grapevine, Texas (a suburb of Dallas-Fort Worth) on Sunday, February 7, 2013. For more information please click here.
This event will be a full day of informative presentations with a focus on basic information about Inflammation Therapy (IT) and remarkable recovery stories. There will be many opportunities to talk with CIR staff, patients on IT and their families.
You're also invited to an informal reception/social with complimentary buffet (gluten-free) and cash bar from 5-8pm Saturday evening - medical practitioners who attended the Physicians' Seminar on Saturday will be in attendance.
We know many of you are on a budget and/or live some distance from Dallas-Fort Worth so we've kept the costs as low as possible. Last year's workshop got rave reviews so we hope you'll return or attend for the first time. We'd love to meet you.
Warm regards,
|
|
Patient Workshop Program | |
7:30 - 8:30 Breakfast Buffet (includes gluten-free)
8:30 - 8:45 Welcome - Belinda Fenter
8:45 - 9:15 Bacterial Etiology of Chronic Inflammation - Kelly Fincher
9:15 - 9:45 Introduction to Inflammation Therapy - Meg Mangin R.N.
9:45 - 10:00 Break
10:00 -10:30 Recovery from Sarcoidosis - Belinda Fenter
10:30 -11:00 Recovery from PCOS - Dr. Barry Rodgers
11:00 -11:30 Recovery from Pemphigoid - Clarence Creacy
11:30 -12:00 Clinicians' Q & A panel - Dr. Barry Rodgers,
Dr.Charles Malis, Dr. Neil Hirschenbein
12:00 - 1:30 Lunch
1:30 - 2:00 Recovery from Hypothyroidism - Debbie Yeager R.N.
2:00 - 2:30 Recovery from Chronic Lyme Disease - Kelly Fincher
2:30 - 3:00 Recovery from Cardiovascular Disease - James Graves
3:00 - 3:15 Break
3:15 - 3:45 Recovery from Fibromyalgia and Diabetes II
Rebecca Sinha
3:45 - 4:30 Round Table discussions:
Family Support & Methods of Coping
4:30 - 5:00 Adjourn and Evaluations
|
|
About Inflammation Therapy | |
Recent epidemiological studies have observed that a high level of 25-hydroxyvitamin (25-D) appears to suppress the inflammatory symptoms of many chronic illnesses. Some have assumed this means 25-D blocks the vitamin D receptor (VDR). This is an incorrect assumption, as shown by researchers in a study published in the Journal of Steroid Biochemistry and Molecular Biology. [1]
How then does 25-D suppress inflammation? Researchers at National Jewish Health have discovered specific molecular and signaling events by which 25-D inhibits inflammation. [2] In their experiments, they showed that low levels of 25-D failed to inhibit the inflammatory cascade, while higher levels inhibited inflammatory signaling.
They observed that two forms of vitamin D (25-D and 1,25-D), dose-dependently inhibited lipopolysaccharide (LPS), a molecule associated with bacterial cell walls that is known to promote intense inflammatory responses.
Cells incubated with no 25-D and in solution containing 15ng/ml of 25-D produced high levels of cytokines IL-6 and TNF-alpha, major actors in the inflammatory response. Cells incubated in 30ng/ml 25-D and above showed significantly reduced response to the LPS. The highest levels of inflammatory inhibition occurred at 50ng/ml.
25-D can also be indirectly immuno-suppressive by being converted to excess 1,25-D. [3] Unfortunately, these researchers believe that suppressing the immune system and thus, inflammatory symptoms, is a good thing despite the fact that it has never been proved vitamin D supplementation reduces risk of disease. [4] However, their findings support the Inflammation Therapy recommendation to maintain 25-D at a level that won't inhibit inflammation because an effective immune system response is essential to eliminate the offending intracellular pathogens which we believe are the root cause of chronic inflammation.
[1] 25-Hydroxyvitamin D3 is an agonistic vitamin D receptor ligand.
Yan-Ru Lou, Ferdinand Molnár, Mikael Peräkylä, Shengjun Qiao, Allan V. Kalueff, René St-Arnaud, Carsten Carlberg, Pentti Tuohimaa
Steroid Biochemistry and Molecular Biology, Volume 118, Issue 3, 15 February 2010, Pages 162-170.
[2] Vitamin D Inhibits Monocyte/Macrophage Proinflammatory Cytokine Production by Targeting MAPK Phosphatase-1.
Yong Zhang, Donald Y. M. Leung, Brittany N. Richers, Yusen Liu, Linda K. Remigio, David W. Riches, And Elena Goleva.
The Journal of Immunology, March 1, 2012.
[3] 1,25-Dihydroxyvitamin D3--a hormone with immunomodulatory properties.
Lemire J.University of California, San Diego Department of Pediatrics, La Jolla CA
Z Rheumatol. 2000;59 Suppl 1:24-7.
[4] Vitamin D for treatment and prevention of infectious diseases: a systematic review of randomized controlled trials.
Yamshchikov AV, Desai NS, Blumberg HM, Ziegler TR, Tangpricha V.
Endocr Pract. 2009;15(5):438-449.
|
|
| |
This study links caffeine consumption to osteoporosis: "In summary, our data provides evidence of a direct effect of caffeine on VDR protein expression and osteoblast activity, which could be one of the probable responsible molecular mechanisms for the role of caffeine in osteoporosis."
Caffeine decreases Vitamin D receptor protein expression and 1,25(OH)2D3 stimulated alkaline phosphatase activity in human osteoblast cells.
"From a clinical and translational significance point-of view, these findings suggest that the use of minocycline offers the advantage of providing both antimicrobial and anti-inflammatory effects, which may be key in treating certain types of infectious diseases, particularly those that lead to hypercytokinemia and chronic inflammatory disorders."
Minocycline modulates cytokine and chemokine production in lipopolysaccharide-stimulated THP-1 monocytic cells.
|
| Quotes | |
"We are going to be moving in February to a new (smaller) home and are involved with all the decisions. It's a lot more activity than I've had in 3 or 4 years, but I'm thrilled to have enough energy to deal with it. Some days I really feel pretty good, although I realize that I still have a long way to go; the mental focus and a bit more stamina are making me feel like I'm coming back to life." Katherine
"I am improving given that I am now only taking pain medication on an occasional basis versus where I have come from. At my peak in pain meds I was taking 2400mg of ibuprofen daily...now only as needed...I am calling that progress!" Flyboy
|
|
|
|
|
