In animal models, increases of circulating endotoxin are correlated with obesity, insulin resistance and diabetes. These results suggest that the chronic, low-grade inflammation associated with diabetes may be a result of bacterial antigens entering the circulation from the intestines. Recently, scientists from the UK demonstrated for the first time that intestinal permeability is indeed increased in humans with diabetes:
Increased Intestinal Permeability to Oral Chromium (51 Cr)-EDTA in Human Type 2 DiabetesScientists at BioDtech, Inc. have successfully removed trace amounts of endotoxin bound to proteins. In addition they have measured previously undetectable levels of endotoxin which could cause activation of the immune system. BioDtech products are designed to more accurately detect endotoxin and remove it from proteins and DNA.
