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| Issue No. 8 |
March 4, 2008 |
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Plant Disease of the Week
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Greetings!
Plant Disease of the Week is a series of online bulletins about plant diseases that affect farms, nurseries, gardens, landscapes and forests in Hawaii. You are welcome to forward this message to anyone who might benefit from the information by using the link at the bottom of the page. You may unsubscribe at any time by using the "SafeUnsubscribe" link at the bottom of this page.
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Late blight of tomato
Phytophthora infestans
Introduction:
The tomato is one of the most widely grown vegetable food crops in the world, second only to the potato. Crops of tomatoes have socioeconomic importance to families, gardeners, farmers, laborers, marketers, retailers, chefs and other workers and services in the food and restaurant industries in Hawaii.
Tomatoes rank as the 10th most valuable agricultural commodity in the state, with a 2005 production value of more than $9.7 million. In addition, there are numerous unaccounted backyard or small tomato gardens in the state, making the tomato plant one of the most important and widely grown food crops.
Yet, a humid and tropical environment favors certain plant diseases. The fact that one lives in the tropics where the climate allows year-round cultivation of tomatoes does not mean it is necessarily a good idea to do so, as many unsuspecting gardeners have learned. A destructive disease, late blight caused by Phytophthora infestans, awaits the tomato where it is cultivated in moist, cool, rainy and humid environs.
This plant pathogen is one of the most notorious and devastating organisms in recent human history, being responsible for the terrible Irish potato famine in the 1840's and is arguably the most important pathogen of potatoes and tomatoes worldwide.
If you planted tomatoes in November and December of 2007 in or near Hilo, Waimea, Kurtistown, Mt. View, Glenwood, Volcano, or Oceanview on the Big Island, or any other areas in Hawaii where cool and rainy and humid conditions prevailed, you may be wondering what happened to your crop. Perhaps you left for a weekend visit on Oahu and you returned after a few days to find your plants virtually destroyed.
Here we treat late blight of tomato and discuss ways to deal with this potentially devastating plant disease.
Disease name: late blight 
Photo: brown, blighted tomato fruits in a garden near Hilo, Hawaii in April 2007, caused by the plant pathogen, Phytophthora infestans (Photo - S. Nelson, UH-CTAHR).
Host (common name): tomato
Host (scientific name): Lycopersicon esculentum L.
Plant family: Solanaceae, the nightshade family
Description of plant: The tomato is a perennial plant with weak, woody, densely hairy stem that often vines over other plants. It reaches 3 - 10 ft in height (1 - 3 m) and bears clusters of edible fruits classified as vegetables.
Geographic range of plant: Native to Central, South and southern North America (Mexico to Peru); now grown in most arable locations globally (either as an indoor or outdoor crop, hydroponically or in soil).
Pathogen: Phytophthora infestans (Mont.) de Bary
Classification of pathogen: Phytophthora infestans is not a true fungus, but rather is regarded as a fungus-like organism. This pathogen is classified as an Oomycete. Oomycetes are members of the kingdom Chromista (= Straminipila). Oomycetes belong to two orders, Saprolegniales and Peronosporales. The order Personosporales contains Phytophthora species and a number of other very important plant-pathogenic genera, including the genus Pythium.
Geographic range of P. infestans: worldwide, but most severe epidemics occur in areas with frequent cool, moist weather.
In Hawaii: Late blight is a common disease of tomatoes at upper elevations in Hawaii where the weather pattern is predominantly cool and moist during periods of the year (winter months). For example, the disease occurs on the Big Island from Hilo to Volcano and even in shadehouse-grown tomatoes in locations such as the Opihikao region of the Big Island near sea level. However, the epidemics tend to coincide with cool and moist winter weather patterns at these locations. Late blight is not as likely to occur in warmer, more arid regions of the state. The other important host for P. infestans is potato. In the early- to mid-1990s, a late blight epidemic struck experimental potato (Solanum tuberosum) plantings at Kunia, Oahu, causing widespread crop loss in a matter of days (S. C. Nelson, unpublished).
Host range of P. infestans: limited to mainly solanaceous crops, including tomato (Lycopersicon esculentum), nightshade, and potato (Solanum tuberosum).
Table 1. Reported plant hosts worldwide of the plant pathogen Phytophthora infestans (after Erwin and Ribeiro, 1996)*.
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Scientific names (Common names) |
Scientific names (Common names) |
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Acer sp. (Maple) |
Mirabilis jalapa L. (Four o-clock) |
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Anthoceris viscosa R. (Anthoceris) |
Nicandra physalodes (L.) Gaertn. (Apple of Peru) |
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Aster thomsonii Clarke (Aster) |
Nicotiana spp. (2 species) (Nicotiana) |
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Atropa belladonna L. (Deadly nightshade) |
Nolana humifusa (Gouan) Johnst. (Nolana) |
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Bupleurum maddeni Clarke (Thoroughwax) |
Petunia spp. (2 species) (Petunia) |
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Capsicum annuum L. (Red pepper) |
Physalis spp. (2 species) (Chinese lantern, tomatillo, ground cherry) |
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Datura spp. (2 species) (Datura) |
Polygonum alatum Buch-Ham (Smartweed, knotweed) |
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Erigeron multicaulis DC (Fleabane) |
Rumex acetosa Linn. (Garden sorrel) |
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Galingsoga parviflora |
Salpichroa origanifolia (Lam.) Baill. (Cock's eggs) |
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Geranium nepalense Sweet (Cranebill) |
Salpiglossus spp. (3 species) (Painted tongue) |
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Hyoscyamus spp. (2 species) (Henbane) |
Schizanthus spp. (3 species) (Butterfly flower) |
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Ipomoea spp. (2 species) (Morning-glory) |
Solanum spp. (47 species) |
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Lycium spp. (4 species) (Matrimony vine) |
Tilia sp. (Lime tree) |
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Lycopersicon esculentum Mill. (tomato) |
Withania somnifera (L.) Dun. |
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Mandragora officinarum L. (Mandrake) |
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*Note on hosts of P. infestans: Host-specific strains or races of P. infestans are common. It is highly unlikely that any given isolate or population of the pathogen, P. infestans, would be capable of infecting and causing disease on all of the hosts listed in Table 1. That a given isolate or population of the pathogen would show decided host preference or be host-specific is much more likely, meaning that in reality the host range of a given isolate or population of P. infestans would be more restricted than indicated by Table 1.
Pathogen variability (strains): There are a number of strains of the P. infestans reported in the United States and worldwide. The strains vary genetically for virulence and have been able to overcome previously resistant plant varieties (potatoes), and also pose the threat of developing resistance to certain types of fungicides.
Sites of infection: all aboveground parts of the plant are susceptible, at any stage of plant development.
Life cycle of P. infestans: Phytophthora infestans is heterothallic (vs. homothallic), having two compatibility mating types designated as A1 and A2. Whenever these two mating types occur together in the same plant tissue, sexual reproduction between the two types produces oospores, otherwise only asexual sporangia are produced. Until the early 1980s, A1 was the predominant mating type recovered from all regions of the world, except central Mexico. A2 isolates are now found in all potato-growing areas of the world. Due to the absence of oospores in samples of tomatoes with late blight in Hawaii examined by the author, it is likely that mating type A1 is responsible for late blight epidemics on tomato in Hawaii.
Summary of the late blight disease cycle:
- Dissemination - sporangia or mycelial fragments are dispersed from infected plant organs by winds and/or splashing raindrops or wind-driven rain.
- Inoculation - sporangia or mycelial fragments land on susceptible host organ(s)
- Infection and pathogen development - sporangia either germinate directly via germ tubes and penetrate a plant organ, or sporangia release motile zoospores which in turn encyst on host organs and penetrate the tissues via a penetration peg.
- Symptom and disease development - mycelium of the pathogen penetrates cell walls directly ramifies intercellularly throughout host tissues, rapidly destroying them and leading to the development of the characteristic necrotic late blight symptoms.
- Reproduction - sexual reproduction is rare in nature; more commonly, asexual reproduction occurs: sporagiophores bearing asexually produced zoosporangia form on diseased tissues at a relative humidity of 91-100% and a temperature range of approximately 3 - 26 C (38 - 79 F) with an optimum temperature between 18 - 22 C (64 - 72 F).
- Survival - Phyophthora infestans survives in plant debris or on volunteer tomato plants and on perennial weeds such as nightshade; where both mating types are present (A1 and A2), P. infestans generates the thick-walled oospores that are longer-term survival propagules.
Pathogen dispersal: wind, rain, or human-assisted (via movement of infested or infected materials such as seed or tools).
Environmental conditions favoring disease development: Daytime temperatures between 60 - 70 F, night temperatures between 50 - 60 F and relative humidity near 100% are the ideal conditions for infection and spread of late blight disease.
Predisposing factors: cool, wet weather and high relative humidity, large, densely planted crops of tomato.
Disease symptoms and signs:
(1) Symptoms on tomato leaves -
Lesions begin as indefinite water-soaked spots that enlarge rapidly into pale green to brownish black lesions, and can cover large areas of the leaf. During wet weather, lesions on the abaxial surface of the leaf may be covered with a gray to white moldy growth (not to be confused with powdery mildew disease). On the undersides of larger lesions, a ring of moldy growth of the pathogen is often visible during humid weather. As disease progresses, the foliage turns yellow and then brown, it curls, shrivels and dies.
Above: Initial symptoms of late blight of tomato on tomato leaves: a rapidly expanding, grayish-brown blight of leaves, petioles and stems. Blighted areas on leaves may have irregular shapes. Disease onset and progression is usually very rapid. Blighted leaves turn yellow and may become curled and deformed within days of becoming infected (Photos - S. Nelson, UH-CTAHR).
Above: Evidence, or signs, of the pathogen (Phytophthora infestans) may be visible as powdery, whitish rings around the margins of the blighted areas. The whitish material consists of mycelia and zoosporangia of the pathogen, Phyophthora infestans (Photos - S. Nelson, UH-CTAHR).
(2) Symptoms on tomato petioles and stems
Lesions begin as indefinite water-soaked spots that enlarge rapidly into brown to black lesions and cover large areas of the leaf. During wet weather, lesions may be covered with a gray to white moldy growth of the pathogen. Affected stems and petioles may eventually collapse at the point of infection, leading to death of all distal parts of the plants.
Above: Elongated, blackened lesions appear on tomato branches and stems. Stems, petioles and leaves may collapse (Photos - S. Nelson, UH-CTAHR).
Above: Elongated, blackened lesions appear on tomato branches and stems. Stems, petioles and leaves may collapse. The pathogen, Phytophthora infestans, may produce a sheen of white mycelium and spores on the surface of these blighted tissues (right) (Photos - S. Nelson, UH-CTAHR).
(3) Symptoms on tomato fruits
Dark, olivaceuos greasy spots develop on green fruit; a thin layer of white mycelium may be present during wet weather.
Above: Infected tomato fruits turn greasy, olivaceous-brown and decay, and can shrivel up and can fall off the plant and never ripen. Such infected fruits are not fit for human consumption. They should be removed from affected gardens and destroyed. The fruit damage that appears in this photograph can occur in a few days or less under the right weather conditions (Photo - S. Nelson, UH-CTAHR).
Crop damage: This disease can spread rapidly during cool, rainy weather, killing plants within a few days and causing total crop loss. Effects on the plant include extensive defoliation; reduced photosynthetic leaf area; loss of plant vigor; plant death; loss of fruits and reproductive capacity, loss of seeds.
Above: A garden planting destroyed by late blight. Virtually all of the tomato plant leaves in a garden can be destroyed within a week's time during weather that is favorable for late blight disease development. Avoid planting tomato plants in large groups where plant diseases have a history of limiting tomato production (Photo - S. Nelson, UH-CTAHR).

Above: (left) A young tomato seedling with leaf, petiole and stem symptoms of late blight disease caused by Phytophthora infestans. The leaf and petioles collapsed, and the stem of the plant is blackened by the disease. The affected plant was growing in a commercial tomato greenhouse near Opihikao on the island of Hawaii in February, 2008; (right) sporangia and sporangiophores of P. infestans (highly magnified). These lemon-shaped sporangia may germinate directly or release swimming zoopores that can infect host tissues. Two of the sporangia in the photograph appear to be transparent, and are indeed empty, having discharged their contents. It is very important to understand that this disease can kill plants in a matter of a few days (photos - S. Nelson, left, and B. Bushe, right, UH-CTAHR).
Other Phytophthora diseases in Hawaii / crops affected by other Phytophthora species. Plant-pathogenic species in the genus Phytophthora affect a wide range of important crops in Hawaii:
Common name of plant (Pathogen scientific name, disease names)
Noni (Phytophthora sp., black flag)
Taro (Phytophthora colocasia, leaf blight)
Pineapple (Phytophthora cinnamomi, Phytophthora parasitica, heart root, root rots)
Papaya (Phytophthora palmivora, fruit blight, stem blight, root rot)
Coconut (Phytophthora katsurae, heart rot)
Potato (Phytophthora infestans, late blight)
Some palm species (Phytophthora sp., root rot)
Citrus (Phytophthora sp. stem canker, root rot)
Avocado (Phytophthora sp. stem canker, root rot)
A'ali'i (Phytophthora sp., root rot)
Some locations in Hawaii where late blight of tomato caused by P. infestans might occur more often (these are examples based on the author's experience, other locations with similar environments are not named and certainly are possibilities):
The Big Island: Hilo, Waimea, Volcano/Mauna Loa Estates, Glenwood, Kurtistown, Waiakea Uka, Puna district, South Hilo district, Kona mauka, Hamakua mauka, Oceanview mauka.
Oahu: Kunia, Kaneohe mauka, anywhere moist and cool
Maui: Kula, Hana, anywhere moist and cool
Historical information and societal impact of P. infestans: Phytophthora infestans was the plant pathogen that destroyed the potato crop in Ireland during the 1840's, causing the Irish potato famine. The great famine had far reaching socio-economic consequences, in particular for Ireland and the United States of America, and for botanical sciences. This pathogen is one of the most intensively analyzed organisms in botany, the study of which allowed Anton de Bary to help spawn the emerging new science of plant pathology in the late 1800's in Europe and the United States.
Integrated pest management practices (IPM):
- The most risky range of months to cultivate tomatoes in blight-prone areas in Hawaii is from November to April. Avoid winter plantings of tomato in areas where prolonged periods of cool, wet, humid weather tend to occur. The pathogen becomes inactive during dry periods. If one is unsure about the climate for a give area, one may check the "Hawaii climate summaries" link in the References to determine if the environment in a particular area tends to favor late blight disease development.
- Select a tomato variety that reaches maturity quickly (i.e., early-bearing or short-season types). This will allow the grower to get the crop harvested as soon as possible. "Early-season" tomato varieties require about 55-67 days to reach maturity; whereas "mid-season" and "late-season" varieties require from about 68-78 days and about 79-85+ days, respectively.
- Grow tomatoes in glasshouses or in spaces where there is humidity control and the plants are covered and protected from rainfall.
- Ensure quick and vigorous tomato seedling growth though adequate and supplemental plant nutrition. Silicate-containing fertilizers may increase pest resistance, especially in the seedling stages.
- Early harvest. If your tomatoes are nearly full size (yet green) and there is a big storm coming and you can use green tomatoes, you might as well harvest them.
- Stake up tomato plants, especially indeterminate types. Keep tomato stems and branches away from the ground.
- Plant blight-resistant tomato varieties when they become available. Late blight-resistant tomatoes are difficult to breed and to find in seed catalogs, and they may not necessarily perform well at your location and may be very susceptible to other diseases. A search of seed company catalogs in February 2008 revealed only two tomato varieties with purported tolerance or resistance to late blight: 'Legend' (determinate, 65-68 days to harvest) and 'Juliet Hybrid' (indeterminate, 60 days to harvest).
- Intercrop tomato with non-susceptible host plants, preferably non-solanaceous plants.
- Disperse the tomato plants around the property and avoid planting them all together in one place. Use wide plant spacing to allow air ventilation of the tomato canopy, which allows wet plant surfaces to dry off most rapidly.
- Crop sanitation - inspect the plants regularly for late blight disease symptoms and promptly remove diseased material from the plot or garden (carefully detach diseased leaves, stems of fruits and destroy them).
- Eliminate cull piles in the vicinity of tomato plantings.
- Use disease-free tomato transplants; inspect seedlings for symptoms and destroy diseased plants promptly. Protect seedlings with fungicide sprays before transplanting.
- Destroy volunteer tomato plants.
- Avoid moving through the tomato garden or field when plant foliage is wet.
- Crop rotation - avoid successive crops of tomatoes in the same location. Avoid planting a new crop of tomatoes beside a diseased crop.
- Do not plant potatoes (S. tuberosum) near tomatoes, or if potatoes are planted, use a blight-resistant variety.
- The plant row orientation should be parallel to the prevailing winds to allow breezes to move between plant rows, which allows more rapid drying of foliage and reduced relative humidity in the plant canopy.
- Irrigation timing - water plants early in day, rather than late in the day. This allows foliage and soil to dry out before evening, lessening the duration of leaf wetness and lowering relative humidity.
- Irrigation method - do not spray foliage with water; keep water off of leaves and stems (irrigate plants at ground level rather than overhead, except for foliar feeding).
- Weed control - control solanaceous weeds around the tomato garden.
- Where possible, apply fungicides on the basis of a weather-based disease forecasting system (see below) rather than on a calendar basis.
- Large growers may decide to use a preventive fungicide spray regime, applying products on a calendar basis. It is best to rotate different products to avoid fungicide resistance.
- Other growers may apply fungicides as soon as symptoms are observed.
- Do not purchase diseased tomato seedlings - inspect them for symptoms first.
- Protect seedlings from nutritional stress and other pests. Potassium silicate fertilizer can help to grow seedlings that are more resistant to fungal diseases and some insect pests.

Above: Solanum nigrum (nightshade) and some other solanceous weeds in the nightshade family are potential hosts for the late blight pathogen, Phytophthora infestans, and should be eradicated from tomato gardens (Photos - S. Nelson, UH-CTAHR).
Buying tomato seeds for disease resistance: Tomato cultivars vary widely in their susceptibility to plant diseases. Generally, contemporary tomato hybrids have better disease resistance than the older heirloom types. Information about disease resistance is coded on the front of tomato seed packages as upper-case letters that signify disease resistance. Look for disease-resistance letters on the seed packages you purchase. The most common letters are: V - Verticillium wilt; F - Fusarium wilt strain I, FF - Fusarium wilt strain I & II, N - Nematodes, T - Tobacco mosaic virus, and A - Alternaria.
Late blight disease forecasting systems:
Proper timing of fungicide sprays conserves resources and provides the best disease control. Late blight epidemics occur within a certain temperature range when the environment becomes very moist and humid. By using accumulated severity values for weather parameters (rainfall, air temperature, relative humidity), fungicide applications can occur at just the right time to protect the plants.
Disease forecasting systems for tomato late blight allow farmers in a region to best time their fungicidal sprays. These computerized forecasting systems use regional weather monitoring stations to make fungicide recommendations for that region. The forecasting systems assume that the pathogen inoculum is probably present and that an epidemic of the disease is highly likely if the right environmental conditions coincide.
The systems predict disease development by the accumulation of various severity values for temperature, rainfall and/or relative humidity, and when a pre-determined threshold of severity values is reached, sprays are triggered. Regional modifications to the original systems have been developed.
1) BLITECAST system - based on temperature, relative humidity and rainfall
A regional disease forecasting system used by potato and tomato farmers in the Northeastern United States. Farmers in a region receive daily updates and spray advisories from their control centers. In the following links are tabular summaries of the BLITECAST values and avisories for three locations in Northeasten Pennsylvania during the 2007 growing season:
2) NoBLIGHT system (Maine), similar to BLITECAST, but differs in the calculation of severity values for relative humidity:
3) HYRE system - based on temperature and rainfall
4) WALLIN system - based on temperature and relative humidity
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Table 2. Some fungicides for sale in Hawaii for year ending December 31, 2008 (for controlling late blight, caused by Phytophthora infestans, on tomato).
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Product name* |
Active ingredient(s) |
Formulation(s) |
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Abound, Quadris, Heritage |
Azoxystrobin (22.9%) |
Flowable concentrate |
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Acrobat 50WP, Forum |
*1 |
Wettable powder |
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Alude, Agfrifos (several products), Fosphite, others |
Mono- and di-potassium salts of phosphorous acid (45.8%) |
Emulsifiable concentrate |
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Amistar |
Azoxystrobin (80%) |
Water dispersible granules |
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Applause 720, Bravo products, Concorde, Initiate 720 |
Chlorothalonil (54%) |
Various |
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Basic Copper 53, |
Basic cupric sulfate (98%) |
Emulsifiable concentrate |
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Bonide copper |
Basic cupric sulfate (7%) |
Wettable powder/dust |
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Cabrio EG |
Pyraclostrobin (20%) |
Emulsifiable concentrate |
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Champ Formula 2 |
Copper hydroxide (37.5%) |
Flowable concentrate |
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Disarm 480 |
*2 |
Flowable concentrate |
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Dithane 75 DF Rainshield
(plus other Dithane products) |
Mancozeb (75%) |
Water dispersible granules, various others |
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Dupont Manex, Griffin Manex |
Maneb (37%) |
Flowable concentrate |
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Dupont Mankocide |
Copper hydroxide (46.1%), Mancozeb (15%) |
Water dispersible granules |
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DuPont Manzate, Griffin Mazate |
Mancozeb (37%) |
Flowable concentrate |
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Dupont Tanos |
Famoxadone (25%); Cymoxanil (25%) |
Water dispersible granules |
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Fluoronil |
Chlorothalonil (72%); Metalaxyl-M (4.4%) |
Water soluble packaging |
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Gavel 75DF |
Mancozeb (66.7%); Zoxamide (8.3%) |
Water dispersible granules |
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Kocide products (several) |
Copper hydroxide |
Various |
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Ranman |
*3 |
Emulsifiable concentrate |
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Ridomil Gold |
Chlorothalonil (72%); Metalaxyl-M (4.5%) |
Wettable powder |
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Serenade |
QST 713 strain of Bacillus subtilis (1.34%) |
Emulsifiable concentrate |
* arbitrary examples of product names; more products may exist with the same active ingredients and/or with different formulations. *1: Morpholine, 3-(3-(4-chlorophenyl)-3-(3,4-dimethoxyphenyl)-1-oxo-2-propenyl (43.1%); *2: Methanone, [2-[[6-(2-chlorophenoxy)-5-fluoro-4-pyrimidinyl]oxy]phenyl] (5,6-dihydro-1,4,2-dioxazin-3-yl)-,O-methyloxime; *3: 1H-Imidazole-1-sulfonamide, 4-chloro-2-cyano-N,N-dimethyl-5-(4-methylphenyl) (34.5%)
METALAXYL. Use of metalaxyl-containing fungicides may pose a particular risk for the development of fungicide resistance in Phytophthora infestans populations, rendering the products potentially ineffective. Resistance to metalaxyl reportedly became a widespread problem when the fungicide was once widely used to control late blight of potatoes in the United States. |
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References
Reference books and journal articles:
(1) Agrios, G. N. 2005. Plant Pathology, Fifth Edition. Elsevier Academic Press, New York. 922 pp.
(2) Ann, P. J., Chang, T. T., and Chern, L. L. 1998. Mating type and pathogenicity of Phytophthora infestans in Taiwan. Bot. Bull. Acad. Sin. 39: 33-37.
(3) Czizinsky, A. A., Schuster, D. J., Jones, J. B., and van Lenteren, J. C. 2005. Crop Protection. pp. 199 - 235 in: E. Heuvelink (ed.) Tomatoes. CABI Publishing, Cambridge, Mass, USA. 339 pp.
(4) Erwin, D. C., and Ribeiro, O. K. 1996. Phytophthora infestans (Mont.) de Bary (1876). pp. 346-353 in: Phytophthora Diseases Worldwide. American Phytopathological Society, St. Paul, Minn., USA. 562 pp.
(5) Integrated Pest Management for Tomatoes, Third Edition. 1990. University of California Statewide Integrated Pest Management Project, Division of Agriculture and Natural Resources, Publication 3274. 105 pp.
(6) Jones, J. B., Jones, J. P., Stall, R. E., and Zitter, T. A., eds. 1991. Late blight. pp. 17-18 in: Compendium of Tomato Diseases. American Phytopathological Society, St. Paul, Minn., USA. 100 pp.
(7) Statistics of Hawaii Agriculture 2005. (December 2006). Hawaii Department of Agriculture and the United States Department of Agriculture. Honolulu, HI.
Acknowledgements: Brian Bushe, UH-CTAHR, for photography; Janice Uchida, UH-CTAHR, for information; Mary Hudak, Hilo, for assistance with photography and for material assistance. | |
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Thank you for reading about this plant disease. Please feel free to contact me if you have any questions or comments about the information provided in this message or if you would like to see a specific plant disease treated in this series.
Dr. Scot C. Nelson
University of Hawaii at Manoa
College of Tropical Agriculture and Human Resources
Department of Plant and Environmental Protection Sciences Cooperative Extension Service
875 Komohana St., Hilo, HI 96720
Note: The information given herein is for educational purposes only. Reference to commercial products or trade names is made with the understanding that no discrimination is intended and that no endorsement by the Cooperative Extension Service is implied. Educational programs conducted by the Hawaii Cooperative Extension Service serve people of all ages regardless of socioeconomic level, race, color, sex, religion, handicap or national origin.
Author: Scot C. Nelson
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Unversity of Hawaii at Manoa
College of Tropical Agriculture and Human Resources
Department of Plant and Environmental Protection Sciences
Cooperative Extension Service
875 Komohana St.
Hilo, Hawaii 96720 tel: 808-981-5199
fax: 808-981-5211 |
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